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steroid user

Testimonial – Triumphs and Tragedies of an Anabolic Steroid User, Part 1

by Austin Knight

Editor’s Note: This is the first installment of a three part series by an anabolic steroid user. In order to give real facts of what happens to users who take steroids, Steroidology feels compelled to give a voice to users who have experienced first hand the positive and negative effects of these drugs.

In this article, I will detail my journey with anabolic steroids and how they have impacted my life. The purpose of this multi-series article is to share my experiences over an 18 year span, in hopes that my experiences become educational. steroid user

I was 18 years old when I realized that my progress at the gym was unsatisfactory. The extent of my research with respect to growth was reading a book and renting some bodybuilding related VHS tapes. The motivation from the little research I had done faded away fairly quickly. It was quite frustrating as I watched several gym goers develop much larger physiques than myself.  “They have to be using steroids.” I said to myself. Without a second thought, I made the decision to find and use steroids.

A few weeks pass by and through networking, I managed to get ahold of a few Boldenone (EQ) vials and Sustanon (a mix of testosterone esters). I was 19 years old at this stage. The seller instructed me to inject 2 CC of each compound once weekly. I asked if I should be worried about any adverse reactions, to which he responded, “No, you’ll be fine”.

I followed his lead and started my first cycle that very evening. Adrenaline, placebo and excitement all fueled my body and mind from the very moment I pulled the needle out of my deltoid. I immediately headed over to the gym for one of my most intense workout sessions ever. I continued with my injection protocol while counting the days and minutes for the day the steroids “kick in.” Disappointment began to settle in around the sixth week when I realized I was not seeing much progress but I still continued with my protocol.

I complained to a few fellow steroid users about my progress and was told that I needed to “eat like a horse.” Following their advice, I started eating anything and everything that came my way. Sure enough, two weeks of eating this way had me showing development and growth. I was excited again and began to eat even more. I grew from a mere 170 lbs to a whopping 195 lbs. I felt great, larger than life and invincible. I did not want to stop my cycle but was told I needed to in order for progress to continue. I was advised to taper off my doses for two weeks. Tapering off the steroids was the “Post Cycle Therapy” ideology at my gym in the mid 90’s.

With my confidence through the roof, I managed to go out on more dates and meet more people. I was never antisocial, but I was on a different level after this cycle. One of those nights while at home with an ex-girlfriend, I stepped out of bed and headed to the restroom. This is when I heard her say “Oh My God”! I thought she was complimenting me, only to find out she was pointing at my back. I turn my head back towards the mirror only to be completely shocked. My back was covered in darkened/brown spots. Some small, some large. I was confused, embarrassed and angry.

post hyperpigmentaion

A back with a very light case of post hyperpigmentation. The spots caused by this skin disorder can be much larger.

I knew it must have had to do with the steroids that I was using. The following day I visited a dermatologist. I did not mention any steroids use in fear of repercussions at the time. I was far from savvy with respect to blood work, but several panels were ordered. The dermatologist was concerned because it suddenly appeared, which is indicative of recent either events or drug intake. It wasn’t too long before I was called in and asked if I used steroids. Apparently my blood work indicated elevated serum testosterone and estradiol. While admitting use is not acceptable in my book today, at the time it seemed appropriate as I wanted a solution for my problem. I was young, and only a solution mattered to me at the time.

The diagnosis that was given to me was Post Hyperpigmentation. This is a condition where acne spots damage the skin, causing it to discolor permanently. While I did experience some acne, it certainly was not as saturated as these darkened spots on my back. Some of which are over 3 inches in diameter. I was not satisfied with the diagnosis and decided to research this on my own. I did however, accept several prescription medications in both forms; oral and transdermal. None of which made a difference.

Over the next year the pigmentation continued to develop, saturating my shoulders and finally stopping at my deltoids. My years of research did not lead to a cure, but certainly indicated that excessive hormonal fluctuation was the cause. Anabolic steroids put my body’s melanin production into overdrive, leaving me with permanent pigmentation. There is no cure. I can only attempt to lighten the spots. This has also proven ineffective as skin lightening does not discriminate to surrounding areas. So while the pigmentation may be slightly lighter, the normal skin is slightly lighter than natural as well. I’m unable to even the ratio.

This is quite frustrating for me. I like to believe that I am a hard worker, a determined weight lifter and have sculpted an above average body. My current statistics are 6.0 ft. tall, 228 lbs at 11% body fat. I’ve been much heavier and much lighter. My current state is quite near to my final goal of 235 lbs at 10% body fat. Unfortunately, I don’t feel comfortable taking my shirt off at the beach, although I am confident (from what I see) that my body shows that harder work has been put in than others roaming the beach.

Sadly, this is the very beginning of my side effects. More issues arise as my body worsens with other imperfections and lifelong defects. Stay tuned for a follow up article as you follow me through this journey.

Part 2

Steroid Books

Testosterone for Life

Increased vitality. More muscle. Improved health. Greater mental agility. These are just a few of the life-enhancing benefits that men with low levels of testosterone can experience when they increase their testosterone level. If you’ve noticed a decrease in your sex drive; experienced erectile dysfunction; or felt tired, depressed, and unmotivated, this authoritative, up-to-date guide from an expert at Harvard Medical School will help you determine if you have low testosterone–a surprisingly common but frequently undiagnosed condition among middle-aged men.

The Testosterone Advantage Plan

Wide shoulders, narrow waist, thick chest, muscular arms and legs: today’s male ideal physique is the same as that of ancient Greece. Aerobics and the Food Pyramid just won’t yield that shape, argues Lou Schuler, certified strength-and-conditioning specialist and fitness director of Men’s Health magazine. For weight loss and the Greek physique, he proposes the “T” (testosterone) plan: weightlifting and a diet of 33 percent each carbs (mostly low-glycemic-index), fat (the “good” kind), and protein; no alcohol; and minimal sweets and processed food. The book includes a meal planner, grocery list, and recipes.

The Steroid Bible

The Steroid Bible also contains comprehensive information on Human Growth Hormone, Insulin, Clenbuterol, and other drugs used by bodybuilders and athletes. In addition, The Steroid Bible lets you in on some of the best kept secrets in bodybuilding by allowing you to examine personal training diaries of top bodybuilders that document drugs used, training routines, diets, and results.

Anabolic Steroids and Making Them

A comprehensive compilation of making anabolic steroids from their original patents. A do it yourself manual.

Anabolic Steroids: Ultimate Research Guide

This Anabolic Steroid Research guide will educate you on every aspect of anabolic steroid use. Whether you are a steroid user and need to know everything about how to use them effectively and safely, or you are a researcher who would like to know every thing there is about anabolic steroids in one book, this is the reference guide for you.

Legal Muscle: Anabolics in America

What’s inside? At well over 400 pages, EVERYTHING anyone could ever want to know about steroids and the law! Read the true details of recent anabolic steroid investigations, arrests and prosecutions from all across America.

Dope: A History of Performance Enhancement in Sports

Since the dawn of athletic competition during the original Olympic Games in Ancient Greece, athletes, as well as their coaches and trainers, have been finding innovative ways to gain an edge on their competition. Some of those performance-enhancement methods have been within the accepted rules while other methods skirt the gray area between being within the rules and not, while still other methods break the established rules. In modern times, doping – the use of performance-enhancing drugs – has been one method athletes and their trainers have used to beat their competition.

The Anabolic Steroid Handbook

The ‘Anabolic Steroid Handbook’ is a reference guide to the world of steroids and related topics. Learn first-hand how to chemically enhance your physique and build an awesome body like the pro’s. The information inside this book can turn a weekend trainer into an iron wielding bodybuilding warrior.

Anabolic Steroids Reference Manual

Anabolics 2007: Anabolic Steroids Reference Manual (Hardcover)

Demystifying Steroids

Demystifying Steroids uncovers the “other side” of anabolic steroids and their possible benefits using real case examples from thousands of patients and medical research. This book empowers the reader to discover a new truth about anabolic steroids for themselves. Dr. Jesse Haggard has organized the information into two sections. The first section is easy to read and discusses steroid use in medicine and sports. Popular beliefs about anabolic steroids are challenged, including the dangers of steroid use.


Steroids (Compact Research Series) (Library Binding)

Steroids and Other Performance-Enhancing Drugs

This brief entry in the new Drugs series argues persuasively against anabolic steroid use. Succinct paragraphs focus on popular and once-respected athletes destroyed by steroids, including examples of anonymous abusers who either succumbed to the fatal effects (by dying themselves or killing others because of “roid rage”) or recovered from their addiction.

Anabolic Steroids – A Question of Muscle

What does it mean when it is simply no longer possible to believe much of the clinical research that is published, or to rely on the judgment of trusted physicians for their prescribing recommendations? Where are we to go and be forewarned of the dangers of these prescribing practices?

Anabolic Steroids and the Athlete

The first edition of this work, published in 1982, concentrated on the athlete’s use of and the physician’s knowledge of, anabolic steroids. This fully updated second edition discusses the continuing controversy over their use in competitive sports.


What are the side effects of steroids?

Section 4

drawing of hothead

Like all drugs, steroids have two types of effects on the body therapeutic effects and non-therapeutic effects.

Taking steroids is known to improve muscle strength and size and give a competitive edge to athletes who train with them. Users are usually impressed with the results of taking the drugs and with their slimmer, more-fit bodies. But there is a downside to taking steroids  side effects that can cause serious damage to many of the bodies functions.

Like all drugs, steroids have two types of effects on the body  therapeutic effects and non-therapeutic effects. Therapeutic effects of steroids are the desired effects of taking steroids  a leaner physic, larger and stronger muscles. Non-therapeutic effects are the unwanted side effects of steroids that can cause damage to the body. Anyone considering using steroids should learn about these side effects and their dangers because many of them can be irreversible, even fatal if the steroids are used improperly.

There are many precautions steroid users can take to minimize the side effects associated with steroid use. Proper dosage, the right physical training, a planned recovery and the use of certain supplements to counteract side effects can make steroid use safer and less harmful.

In fact, there is some debate to just how harmful steroids are when properly used. Most studies on steroids side effects were conducted at a time when users were unaware of their dangers and tended to take much larger amounts of the drugs than were needed and supplements to counteract the side effects weren’t yet developed. Also, the steroids themselves were not as advanced as ones available today. According to Jerald Bain of the University of Toronto, many of the side effects associated with steroids, like liver damage and heart problems, are related to the classical anabolic agents 17-alkylated steroids. He points out, however, that these steroids are not pure testosterone, the basis for steroids, and that increased levels of testosterone in the body show no signs of causing these harmful side effects, and may in fact be helpful to the body.

As the science of steroids improves, there may be a time when their use will be safe. But for now, their side effects should be looked upon with serious consideration before using any kind of steroid. The following are some of the known side effects of steroids:

Liver damage Steroids contain toxins that the liver cleanses from the body, which may lead to cysts or tumors.

Kidney damage Kidneys also help cleanse the body of toxins, and although it is rare, steroid use may lead to kidney damage.

Cardiovascular problems Steroids affect the cardiovascular system in many ways. Steroids can raise cholesterol levels in the body, which can lead to blocked arteries around the heart or blood clots throughout the body, including the brain. Also, steroid use can lead to cardiomyopathy where the heart muscle grows larger and weaker and is unable to pump blood throughout the body. Finally, steroid use can lead to high blood pressure, which is the leading cause of heart attacks and strokes.

Problems with the reproductive system Steroid use affect the reproductive systems of both men and women. In men, high doses of steroid lead to infertility while they are using steroids. Also, steroid use may cause men to develop female characteristics such as loss of body hair, decreased size of testicles and they may also develop larger breasts. In women, the use of steroids may lead to male characteristics like facial hair and loss of scalp hair. Steroids also alter a woman’s menstrual cycle and make them unable to become pregnant while using steroids.

Stunted skeletal growth Testosterone is the body’s chemical messenger that slows growth of bones during puberty. Adolescents who use steroids can seriously stunt their growth and become less developed than they would be without using steroids.

Skin problems The toxins in steroids when excreted through glands in the skin cause users to suffer from acne.

Psychological problems Steroid use can affect peoples moods in a variety of ways. Some users report a high from taking steroids, which is often followed by a low. Long-term use without the proper recovery plan can lead to serious depression. Also, many users report heightened feelings of aggression, a state that is now coined as roid rage.

Who are using Steroids today

Tutorials Section 2

Who are using steroids today and why?

picture of anabolic bodies

Some of these steroid users include military professionals, law enforcement officers, bouncers or any other person whose job requires a good deal of strength.

Steroid writer Lena Butler reports that recent polling among steroid users suggest that almost 80 percent of steroid users are body builders who don’t compete or play any sports whatsoever. And most are young men in their mid to late 20s, with middle-class backgrounds. Some of these steroid users include military professionals, law enforcement officers, nightclub bouncers or any other person whose job requires a good deal of strength. But an increasing majority of steroid users today are business professionals who cite weight loss, shaping up their bodies and reversing aging as reasons for taking the drugs.

The influence on high school students by professional athletes taking steroids and succeeding to become super stars who make millions of dollars has become a real concern in today’s world of Congressional hearings on doping in baseball and gold medals stripped by the Olympic committee and for good reason. Students who use steroids can cause irreparable damage to their health and their numbers are on the rise.

According to the Child Trends Data Bank, steroid use among eighth and tenth graders in the 1990s stayed at around 1 percent. For tenth graders, that number has more than doubled in this decade. The majority of high school steroid users are male athletes. Over five percent of high school seniors who competed in sports reported using steroids at least once during their years in school. A little over two percent of seniors reported using steroids and did not belong to a sports program. Much like steroid use among professional athletes, little is known about the number of collegiate athletes who use steroids because of rules banning their use. It is surveyed, however, that only about one percent of non-athletes in college use steroids during their time in school.

Although not as publicized as the scandalous professional athlete steroid user, or as mundane as the bouncer/amateur weightlifter steroid user or as scary as the student athlete steroid user, there is a sub sect of the steroid culture that has a very positive story about its use  those who take steroids for medical reasons. Steroids are often administered by doctors to patients suffering from cancer, HIV/AIDS, or any other disease that may cause the muscles to atrophy. Patients taking steroids report greater mobility and an easier time exercising which helps their recovery.

While the steroid using athletes who we watch nightly on ESPN may take up a lion’s share of the attention about steroid use, it is important to remember that there are many others who use steroids for a variety of reasons and to varying degrees.


Heavy Resistance Exercise Lowers Androgen Receptor Levels

picture of resistence_training

The results were a bit surprising in light of some
previous studies, and suggest a possible supplement regimen to offset some of the negative effects that were observed.

Ratamess and coworkers recently published the results of a study that looked at the hormonal profile and androgen receptor content in the vastus lateralis
muscle (a portion of the muscles comprising the quadriceps) of men following two exercise protocols [1]. The results were a bit surprising in light of some
previous studies, and suggest a possible supplement regimen to offset some of the negative effects that were observed. The salient results of their research were (a) an increase in both cortisol and testosterone levels after multiple sets of squats; and (b) a significant downregulation of the androgen receptor in biopsied muscle tissue.

Several previous studies have examined hormonal changes in cortisol,
testosterone, and growth hormone (GH) during and following resistance exercise [2-4]. In [2] Kraemer et.al. observed an increase in both testosterone and GH after heavy resistance exercise. Hakkinen and Pakarinen observed increases in free and total testosterone, cortisol, and GH after an acute bout of heavy squatting [3]. Kraemer et.al. examined plasma hormone changes after an intense bout of cycling and noted a significant increase in cortisol [4]. The current study and the earlier ones cited show a trend of increased cortisol and testosterone immediately after strenuous exercise.

The current study by Ratamass et.al. is the first to look at androgen receptor
content in worked muscle immediately post-exercise. While the elevated
testosterone that many studies show occurs after exercise sounds beneficial, if receptor levels are low, then the increased testosterone would be of less
anabolic value than if receptor levels were unchanged or increased. In fact, a
depressed level of AR is exactly what Ratamass and coworkers found. The
downregulation of AR coupled with high cortisol levels post-exercise would be
expected to make for a metabolic state characterized by net catabolism.

To quote from the current study under investigation,

“…acute hormonal elevations are without context unless subsequent interaction
with a specific membrane bound or nuclear receptor occurs and the appropriate signal is transduced”.

In other words, what good is the extra testosterone produced during lifting if
the receptors aren’t there to accept it?

In the current study, 9 young resistance trained men performed two exercise
protocols. One consisted of a single set (SS) of 10 reps of heavy squats. The
second exercise involved 6 sets of 10 reps of squats (MS). Weights were
determined for each individual by measuring their 1 Rep Max (RM) and then having them squat at 80 – 85% of the (RM). The average RM was 330.4 lbs.

Plasma testosterone and cortisol were measured every 15 minutes for 1 hour after both sessions. The vastus lateralis was biopsied to determine AR content 1 hour after training. The results, taken from [1] are shown below.

As can be seen, there was no significant change in cortisol in is SS group,
while cortisol rose about 40% in the MS group after 30 minutes

Similarly, testosterone did not change in the SS group but showed a transient
increase of 20% in the MS group.

The bar graph below from [1] shows relative vastus lateralis AR content at
baseline and 1 hour after completion of exercise. The drop in AR content in the worked muscle is clear

The authors of the present study attribute the decline in androgen receptors to an overall loss of protein due to the demands of strenuous exercise. Cortisol is highly catabolic to proteins and does not discriminate between contractile proteins and noncontractile proteins, such as the androgen receptor, which itself is a protein. A number of studies have shown that the AR is upregulated after a longer post exercise time period. For example, Bamman & Shipp reported that in humans AR messenger RNA in the vastus lateralis increased 63% and 102% respectively 48 hours following 8 sets of 8 reps of either eccentric (110% of 1 RM) or concentric ( 85% of 1RM) squats [5]. Thus resistance exercise may ultimately upregulate the AR, but the initial response appears to be a catabolic one, based on the current study.

One might be tempted to speculate the increased testosterone and decreased AR may cancel each other out. This may not be the case. Another interesting finding of this study was the individual baseline 1 RM was independent of plasma testosterone levels, but correlated highly with androgen receptor content. So an individual’s AR levels may be more indicative of their strength that their testosterone levels.

Certain anabolic steroids such as Anavar (oxandrolone), that are considered to have a very high anabolic to androgenic ratio are noted for their ability to
upregulate the AR [6].

Since it is generally believed that protein synthesis peaks in the few hours
after a training session, it makes sense to attempt to limit the downregulation
of the AR that seems to occur after exercise. One strategy might be to
supplement with amino acids, especially Branched Chain Amino Acids rich in
leucine. Besides being anabolic in and of itself, leucine taken as a supplement
will be preferentially oxidized for fuel, sparing body proteins, which would
likely include the AR:

The Ergogenic Effects of Citrulline Malate combined with Branched Chain Amino Acids

Another strategy would be to combine a cortisol blocker such as 7-oxo DHEA
and/or phosphatidyl serine to the BCAA mix to help limit protein catabolism.
While I don’t advocate the use of anabolic steroids, clearly agents such as
Anavar which upregulate the AR would likely prove helpful as well.

While elevated cortisol is a likely contributor to protein catabolism, other
proteolytic mechanisms may be at work as well. The body has three independent systems for degrading and disposing of proteins. These are the so-called lysosomal and calcium mediated proteases, and the ATP-ubiquitin dependent proteolytic pathway. However, cortisol has been implicated in activting the ATP-ubiquitin proteolytic pathway [7], which may ultimately be the mechanism by which cortisol exerts its catabolic action; so here again cortisol blockers might help.

We mentioned Anavar above. Besides upregulating the AR, Anavar also antagonizes the catabolic actions of cortisol [8]. Calcium mediated proteolysis is suppressed by cyclic adenosine monophosphate (cAMP), and forskolin is well know to elevate cAMP. Thus forskolin may be a worthwhile supplement to defend against this pathway of protein breakdown. Beta adrenergic agonists, either synthetic such as Clenbuterol or albuterol, or naturally occurring epinephrine and norepinephrine also elevate cAMP and suppress calcium mediated protein breakdown [9]. Ephedrine elevates cAMP directly by binding to beta receptors, and indirectly by increasing levels of the body’s naturally occurring hormone/neurotransmitter norepinephrine.

Newly published research also shows that Clenbuterol, besides inhibiting calcium dependent proteolysis, also acts to block ATP-ubiquitin mediated protein breakdown [10].

Finally, both the lysosomal breakdown of protein and the ATP-ubiquitin
proteolytic system are suppressed by insulin [11,12], so adequate carbohydrate intake prior to, during and after strenuous exercise should help blunt these pathways of protein breakdown.

Thus we have several strategies for reducing the breakdown of androgen receptor proteins after exercise, some as simple as eating to elevate insulin, as well as perhaps even increasing those receptor numbers with the use of certain anabolic steroids such as oxandrolone.


MR, French DN, Vescovi JD, Silvestre R, Hatfield DL, Fleck SJ, Deschenes MR.
Androgen receptor content following heavy resistance exercise in men. J Steroid
Biochem Mol Biol. 2005 Jan;93(1):35-42.

Kraemer WJ, Gordon SE, Fleck SJ, Marchitelli LJ, Mello R, Dziados JE, Friedl K,
Harman E, Maresh C, Fry AC. Endogenous anabolic hormonal and growth factor
responses to heavy resistance exercise in males and females. Int J Sports Med.
1991 Apr;12(2):228-35.

Hakkinen K, Pakarinen A. Acute hormonal responses to two different fatiguing
heavy-resistance protocols in male athletes. J Appl Physiol. 1993

Kraemer WJ, Patton JF, Knuttgen HG, Marchitelli LJ, Cruthirds C, Damokosh A,
Harman E, Frykman P, Dziados JE. Hypothalamic-pituitary-adrenal responses to
short-duration high-intensity cycle exercise. J Appl Physiol. 1989


Nolva vs. Clomid for PCT

picture of nolva_clomid

This misconception originates from their
completely different uses

by liftsiron

It seems like everyday questions concerning PCT pop up, and weather one should
use either Clomid or nolva or a combo of both. I hope that this article written
by BigCat may help to clear up some misconceptions.

While practically similar compounds in structure, few people ever really consider
Clomid and nolva to be similar. Its not just a common myth in steroid circles,
but even in the medical community. This misconception originates from their
completely different uses. Nolvadex is most commonly used for the treatment
of breast cancer in women, while Clomid is generally considered a fertility
aid. In bodybuilding circles, from day one, Clomid has generally been used as
post-cycle therapy and Nolvadex as an anti-estrogen.

But as I intend to demonstrate this is in essence the same. I believe the myth
to have originated because nolva is clearly a more powerful anti-estrogen, and
the people selling Clomid needed another angle to sell the stuff, so it was
mostly used as a post-cycle aid. But few users really understand how Clomid
(and also Nolvadex, logically) works to bring back natural testosterone in the
body after the conclusion of a cycle of androgenic anabolic steroids. After
a cycle is over, the level of androgens in the body drop drastically. The body
compensates with an overproduction of estrogen to keep steroid levels up. Estrogen
as well inhibits the production of natural testosterone, and in the period between
the return of natural testosterone and the end of a cycle, a lot of mass is
lost. So its in everybody’s best interest to bring back natural test as soon
as humanly possible. Clomid and Nolvadex will reduce the post-cycle estrogen,
so that a steroid deficiency is constated and the hypothalamus is stimulated
to regenerate natural testosterone production in the body. That’s basically
how the mechanism works, nothing more, nothing less.

Both compounds are structurally alike, classified as triphenylethylenes. Nolvadex
is clearly the stronger component of the two as it can achieve better results
in decreasing overall estrogen with 20-40 mg a day, than Clomid can in doses
of 100-150 mg a day. A noteworthy difference. Triphenylethylenes are very mild
estrogens that do not exert a lot, if any activity at the estrogen receptor,
but are still highly attracted to it. As such they will occupy the receptor
and keep it from binding estrogens. This means they do not actively work to
reduce estrogen in the body like Proviron, Viratase or arimidex would (by competing
for the aromatase enzyme), but that it blocks the receptor so that any estrogen
in the body is basically inert, because it has no receptor to bind to.

This has advantages and disadvantages. The disadvantage is that when use is
discontinued, the estrogen level is still the same and new problems will develop
much sooner. The advantage is that it works much faster and has results sooner
than with an aromatase blocker like Proviron or arimidex. Therefor, when problems
such as gynocomastia occur during a cycle of steroids one will usually start
20 mg/day of nolva or 100 mg/day of Clomid straight away, in conjunction with
some Proviron or arimidex. The proviron or arimidex will actively reduce estrogen
while the Clomid or Nolvadex will solve your ongoing problem straight away.
This way, when use is discontinued there is no immediate rebound.

So which one should you use? Well personally, I’d have to say Nolvadex. Both
as an on-cycle anti-estrogen and a post-cycle therapy. As an anti-estrogen its
simply much stronger, demonstrated by the fact that better results are obtained
with 20-40 mg than with 100-150 mg of Clomid. For post-cycle, this plays a key
role as well. It deactivates rebound estrogen much faster and more effective.
But most importantly, Nolvadex has a direct influence on bringing back natural
testosterone, where as Clomid may actually have a slight negative influence.
The reason being that tamoxifen (as in Nolvadex) seems to increase the responsiveness
of LH (luteinizing hormone) to GnRH (gonadtropin releasing hormone), whereas
Clomid seems to decrease the responsiveness a bit1.

Another noteworthy fact about Nolvadex is that it acts more potently as an
estrogen in the liver. As you remember, I mentioned that clomiphene and tamoxifen
are basically weak estrogens. Well, tamoxifen is apparently still quite potent
in the liver. This offers us the positive benefits of this hormone in the liver,
while avoiding its negative effects elsewhere in the body. As such Nolvadex
can have a very positive impact on negative cholesterol levels2 in the body,
and therefore too should be considered a better choice than Clomid. It will
not solve the problem of bad cholesterol levels during Steroid use, but will
help to contain the problem to a larger degree.

Another reason why I promote the use of Nolvadex over Clomid post-cycle (as
if being 3-4 times stronger and having more of a direct effect on restoring
natural test wasn’t enough) is because it’s a lot safer. Not just because it
improves lipid profiles, but also because it simply doesn’t have the intrinsic
side-effects that Clomid has. Clomid causes more acne for sure, but that’s mainly
because you need to use a 3-4 times higher dose. But Clomid seems to also affect
the eyesight. Long-term Clomid therapy causes irreversible changes in eyesight3
in users. Irreversible. For me that alone is reason enough to prefer Nolvadex.

Lastly, one should be aware that use of these compounds can reduce the gains
made on steroids. Nolvadex more so than Clomid, simply because it is stronger.
Estrogen is responsible for a number of anabolic factors such as increasing
growth hormone output, upgrading the androgen receptor and improving glucose
utilization. This is why aromatizing steroids like testosterone are still best
suited for maximum muscle gain. When reducing the estrogen levels, we therefore
reduce the potential gains being made. For this reason one may opt to try Clomid
during a cycle instead of Nolvadex. Although I would imagine that the problem
that needed solved would be of more concern, in which case nolva remains the
weapon of choice. It’s a plain fact that there is a high correlation between
gains and side-effects. Either you go for maximum gains and tolerate the side-effects,
or you reduce the side-effects, and with it the gains. That’s life, nothing
is free.

Stacking and Use:

If problems of Gynocomastia or other estrogen related symptoms tend to pop
up during a cycle the use of 20-30 mg of Nolvadex or 100 mg of Clomid daily
should easily contain the problem, and be used until a few days after the problem
subsides. For best results and the least amount of problems upon cessation it
is best stacked with Proviron (50 mg) or arimidex (0.5 mg) for this duration
as well. Its not advised that these products be ran concomitantly with the steroid
for the entire duration of the stack, as this will reduce your gains. Instead
cease the usage of anti-estrogens once the problem is contained, and should
the problem resurface, simply recommence the use of the products in the same
manner as described above.

Once a cycle of steroids is concluded one should always initiate a post-cycle
therapy to help bring back natural testosterone as soon as possible. This will
help you to retain the mass you gained. How this is done depends highly on the
type of steroid used. If only orals were used, therapy should start immediately,
even the last day of the stack. If short-acting esters or water-based injectables
were used, therapy should commence within 4-7 days after last injection, and
if long-acting esters were used then it should commence 1.5 to 2 weeks after
the last injection was given. The length of the therapy will vary as well, from
3-5 weeks. The longer acting the product was, the longer therapy should be continued
to make sure all suppressive factors are cleared before use of Clomid/Nolvadex
is discontinued.

For best results, it is best stacked with HCG (Human Chorionic gonadotrophin),
which functions as an LH analog and can help bring testicle size back up. HCG
use starts the last week of a cycle, and on from there every 5-6 days (usually
1500-3000 IU) and discontinued 1.5 to weeks prior to the cessation of Nolvadex/clomid.
The reason being that HCG itself is also suppressive of natural testosterone
and should be out of the body before therapy is over, or it will inhibit natural
testicle function. But I can not stress enough that HCG possibly plays a more
important role in post-cycle therapy than clomid/Nolvadex. For Clomid and Nolvadex,
doses are usually tapered down. Its best to start with 40-50 mg of Nolvadex
or 150 mg of Clomid for the first week or the first two weeks, and then finish
the program with 20-25 mg of Nolvadex or 100 mg of Clomid for an additional
two weeks.


Aromasin (exemestane)

picture of aromasin

Developed to help fight breast cancer, Aromasin is one of the most powerful estrogen suppressing compounds


6-Methylenandrosta-1,4-diene-3,17-dione; 10,13-Dimethyl-6-methylidene-7,8,9,10,11,12,13,14,15,16-decahydrocyclopenta[a]phenanthrene-

Molecular Formula C20H24O2
Molecular Weight 296.40
CAS Registry Number 107868-30-4
Melting point 155.13 ºC

Developed to help fight breast cancer, Aromasin is one of the most powerful estrogen suppressing compounds available for body builders taking anabolic steroids. Additionally, it also raises testosterone in the body, which is a bonus for bodybuilders during post-cycle-therapy.